Thus, it truly is conceivable that the JIP1/three → ASK1 → MKK4 signaling module might function a conduit to activate JNK when endosomal trafficking is disrupted and glucose levels fall during the early levels of methuosis. Our discovering that MKK4 was robustly activated from the methuosis-inducing IPPs, MOMIPP and 2q, https://johnn777lev8.therainblog.com/profile