Several mitochondrial defects have been located in the mouse model of Pompe condition, together with mitochondrial calcium excess, greater reactive oxygen species, decreased mitochondrial membrane prospective, and lessened oxygen use and ATP output (Lim et al (2015) A murine Niemann‐Choose C1 I1061T knock‐in design recapitulates the pathological characteristics of the https://eleanorh925nkg6.tkzblog.com/profile