Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. Conversely, our ability to rescue SERT perform in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by greater pFAK localization in five-HT synapses. Our data https://fridaw975yjv6.blogitright.com/profile